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Despite major advances in the field concerning the neurobiological substrates underlying the ES-induced increased risks for adverse neurobehavioral outcomes and psychopathology, the underlying mechanisms remain complex, multi-faceted, not yet completely understood and often hard to target, rendering the development of effective interventions challenging [8, 9]
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Genetic disorders of Vitamin D biosynthesis and degradation
[] In rat models, NAC and deferoxamine prevent protein deterioration in the PFC and hippocampal regions
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